Abstrak: Tingkat kebisingan di Instalasi Pengolahan Air (IPA) cukup tinggi. Meningkatnya kebutuhan air bersih seiring dengan bertambahnya populasi penduduk, membuat Perusahaan Daerah Air Minum (PDAM) dituntut untuk meningkatkan kapasitas produksi. Terdapat alat-alat dan proses produksi yang memiliki karakteristik berbeda dibanding jenis industri lain. Terdapat 306 PDAM di seluruh Indonesia, potensi jumlah pekerja yang terpajan bising sangat besar, maka perlu diteliti lebih lanjut mengenai hubungan karakteristik bising serta faktor-faktor determinannya terhadap gangguan pendengaran pada pekerja di PDAM untuk memperoleh bentuk pengendalian yang paling tepat. Penelitian ini menggunakan desain studi potong lintang. Tahapan penelitian ini yaitu mengukur tingkat kebisingan serta memberikan kuesioner sebagai data primer, menganalisis hasil audiometri pekerja sebagai data sekunder dan menggunakan uji statistika Chi Square dan analisis multi determinan untuk mengetahui hubungan di antara variabel independen dan dependen. Hasil penelitian diperoleh bahwa sumber bising di instalasi pengolahan air adalah pompa, exhaust fan, kompresor, blower, vacuum dan terjunan air. Sebanyak 84.4% pekerja di area produksi terpajan bising > 85 dBA. Sebanyak 15.6% pekerja mengalami gangguan pendengaran. Diperoleh kesimpulan bahwa pekerja yang terpajan bising di atas 85 dBA yang memiliki frekuensi bising dominan > 2000 Hz dapat menyebabkan terjadinya gangguan fungsi pendengaran dan diperparah apabila pekerja berusia > 40 tahun dan memiliki masa kerja > 14 tahun. Kata kunci: Kebisingan, PDAM, Gangguan pendengaran, Frekuensi bising

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Noise level in Water Treatment Plant (WTP) is high enough. Increasing the need for clean water in line with the increasing population, making the Water Supply Company (PDAM) is required to increase production capacity. There are machines and production processes that have different characteristics than other types of industries. There are 306 PDAMs throughout Indonesia, the potential number of workers exposed to noise is very large, it is necessary to further investigate the relationship between noise characteristics and its determinants to hearing loss to PDAM workers to obtain the most appropriate form of control. This study used a cross sectional study design. The stages of this study are to measure the noise level and provide questionnaires as primary data, analyzing the worker audiometric results as secondary data and using Chi Square statistical test and multi determinant analysis to find out the relationship between independent and dependent variables. The results obtained that the source of noise in water treatment plants are pumps, exhaust fan, compressor, blower, vacuum and waterfall. About 84.4% of workers in the production area exposed to noise > 85 dBA. About 15.6% of workers have hearing loss. It is concluded that exposure workers over 85 dBA with dominant noise frequency > 2000 Hz can cause hearing impairment and aggravate if workers are > 40 years old and have a working life > 14 years. Key words: Noise, Water supply company, Hearing loss, Noise frequency

Abstrak:

Pekelja di kilang minyak tcrutama di lokasi bising mempunyai risiko tinggi menderita gangguan pcndcngaran sebagai penyakit akibat kelja. Pajanan kombinasi (bising, penyakit DM, hipcrtensi, DM dan hipertensi) dapat terjadi secara bersamaan pada seorang pekerja. Penelitian bertujuan untuk mengetahui hubungau dan kontribusi pajanan kombinasi terhadap teljadinya gangguan pendengaran. Dengan metode historikal kohor, data pekelja dari tahun 2002-2007, pcfnetapan kriteria gangguan pendengaran berdasarkan hasil audiogram pada frekuensi 4000 Hz > 25 dBA, status DM berdasarkan kriteria diagnostik PERKENI 2006 GDP 2126 mg/dL, status hipertensi herdasarkan JNC7 S 2140 mmHg dan D 290 mm!-Ig. Analisis statistik dengan univariat dan bivariat. Diperoleh hasil, insiden gangguan pendengaran berkisar antara 25.0% - 50.0% pada pekelja yang terpzgan bising, dengan distribusi responden menurut masing- masing pajanan kombinasi dan karakteristik responden (variabcl pcrancu: Lunur; rnasa kerja; merokok; dan pemakaian APT). Kesimpulan yang diperoleh dari uji statistik, baik variabel independen maupun vadabel perancu mempunyai p value > on, perbedaan tidak bermal-ma. Sehingga gambaran dan kontribusi faktor risiko penyakit DM dan hipcrtcnsi bclum dapat diketahui dengan jelas. Hal ini disebabkan beberapa keterbatasan penelitian diantaranya sampel yang mcmenuhi kriteria inklusi (140 responden) tidak memenuhi besar sampel minimal (287 responden}, informasi data yang diperoleh dari perusahaan tidak lengkap. Oleh karenanya saran bagi pemsahaan agar lebih memperhatikan sistem pencatatan, pelaporan, dan penyimpanan data., pemeriksaan audiometri, kesehatan berkala, pengukuran dosis pajanan, secara rutin dan berkcsinarnbungan sesuai kebutuhan, terulama bagi pekerja yang terpajan bising > 85 dBA, penertiban sertifikasi operator, kalibrasi alat oleh institusi yang bezwenang.

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Workers of refinery in noisy area have high risk to get hearing loss as occupation disease. Combined exposure (noise, DM, hypertension) can happen simultaneously on a worker. The purpose of this study is to find the relation and contribution of combined exposure on hearing loss. The study was using historical cohort, worker’s data from 2002 to 2007, hearing loss criteria definition based on audiograrn result with frequency 4000 Hz >25 dBA; DM status based on PERKENPS diagnosis in 2006 GDP 2 126 mg/dL, hypertension status based on JNC7 S 2 |20 mml-lg and D 2 90 mml-Ig. Statistical analysis was using univariat and bivariat. The result is hearing loss incident on workers exposed by noise around 25.0% - 50.0%, with respondent’s distribution based on each combined exposure and respondent's characteristic (confounding variable : age, working period, smokind and the using of APT) We conclude by statiscal test, both independent variable and cofounding variable with P value > ot that there is insignificant dillerencetherefore, the illustration and contribution of DM and hypertension risk factor cannot be found clearly. It was caused by some limitations in the study such as inclusive criteria sample (140 respondents) didn't iillfil the quota of sample (287 respondents), incompleted company's clatas. Therefore, we suggest that the company should pay more attention to data entry, data report and data saving, audiometric check-up, periodic medical check-up, exposure dosage measurement, regularly and continually based on needs, especially for workers exposed by noise > 85 dBA, regulation of operator certification, calibrated equipment by authorized institution.

Abstrak:

Gangguan pendengaran karena bising merupakan salah satu penyakit akibat kerja yang sering ditemui pada perusahaan manufaktur. Hazard yang bisa menyebabkan gangguan pendengaran meliputi bising, zat kimia dan getaran. Ruang lingkup penelitian tesis ini adalah melihat dampak pajanan bising terhadap fungsi pendengaran pekerja yang terpajan bising diatas 82 dBA. Jenis penelitian adalah cross sectional study yang meneliti hubungan faktor independen berupa dosisi pajanan dalam perhitungan leq, umur dan masa kerja serta faktor penggangu berupa pemakaian alat pelindung diri serta kebiasaan dengan fungsi pendengaran pekerja. Dari survei tingkat bising ditemukan departemen PVC, CDM, CDS dan CDB mempunyai tingkat kebisingan diatas nilai ambang batas yang diperbolehkan.

Hasil pemeriksaan audiometri ditemukan dua orang responden yang mengalami gangguan pendengaran. Responden yang mengalami gangguan pendengaran satu orang berumur diatas 40 tahun, bekerja pada ruangan PVC dimana merupakan tingkat pajanan bising tertinggi di pabrik ini dan sudah bekerja selama lebih dari 5 tahun. Responden yang mengalami gangguan pendengaran lainnya merupakan pekerja yang berumur dibawah 40 tahun dan sudah bekerja selama lebih dari 5 tahun. Dari hasil analisis statistik tidak ditemukan hubungan yang signifikan antara Leq pajanan bising, faktor masa kerja, pemakaian alat pelindung diri dan kebiasaan merokok dengan gangguan pendengaran. Ditemukan hubungan yang signifikan antara umur dan gangguan pendengaran dengan OD ratio 7.99.

Noise induced hearing loss is one of the occupational diseases are often found in manufacturing companies. Hazard that can cause hearing loss include noise, chemicals and vibration. The scope of this thesis research on the impact of noise exposure on hearing function of workers exposed to noise above 82 dBA. This type of research is a cross-sectional study examining the relationship be an independent factor in the noise dose exposure (leq), age and working period and disturbance factors such as the use of personal protective equipment, smoking with hearing function. From the survey found noise levels PVC department, CDM, CDS and CDB have noise levels above the permitted threshold value.

Audiometric examination found two participant who suffered from hearing loss. Respondents who suffered from hearing loss a person aged over 40 years, working on PVC indoor noise exposure level which is the highest in the plant and it has been working for more than 5 years. Other participant who suffered from hearing loss is under the age of 40 years and has been working for more than 5 years. From the analysis found no statistically significant relationship between Leq noise exposure, working period, the use of personal protective equipment and smoking with hearing loss. Found a significant relationship between age and hearing loss with OD ratio 7.99.

Abstrak: Paparan kebisingan merupakan penyebab paling umum gangguan pendengaran, menyebabkan noise induced hearing loss (NIHL). Penelitian ini mengevaluasi gangguan pendengaran yang berhubungan dengan pajanan bising dikaitkan dengan usia, masa kerja, lama pajanan, pemakaian alat pelindung diri, kebiasaan merokok, hobi berhubungan kebisingan dan penyakit Diabetes Mellitus, hyperlipidemia dan hipertensi pada pekerja. Ini adalah penelitian observational cross sectional meneliti variabel independen, variabel dependen dan variabel perancu pada waktu bersamaan. Menggunakan data sekunder perusahaan melalui pengamatan, pengukuran dan questioner. Hasil pengukuran kebisingan area berpotensi kebisingan menunjukan potensi kebisingan terendah adalah 63 dBA dan tertinggi 110, 6 dBA,tingkat kebisingan area field berkisar 84.88 - 93 dBA. Kebisingan di area nonfield tertinggi 79.5 dBA. Pajanan bising efektif di bawah 80 dBA, baik di area field maupun nonfield; 7.1% pekerja bekerja > 20 tahun, didapatkan hubungan antara masa kerja > 20 tahun, terjadinya gangguan pendengaran pekerja sebanyak 5.6%, 40.5% pekerja berusia > 40 tahun, didapatkan hubungan antara usia pekerja dengan kejadian gangguan pendengaran. 42.9% pekerja memiliki kebiasaan merokok, tidak didapatkan hubungan antara perilaku merokok dengan gangguan pendengaran. Tingkat pemakaian APT pada pekerja didapatkan sebanyak 90.5% pekerja yang selalu memakai APT, tidak ada hubungan antara pemakaian APT dengan gangguan pendengaran. Tidak didapatkan hubungan antara hobi dengan terjadinya gangguan pendengaran Tidak didapatkan hubungan antara status kesehatan berupa profil lipid pekerja (kolesterol total, HDL, LDL, dan trigliserida), kadar glukosa darah pekerja dan tekanan darah dengan gangguan pendengaran.
Kata Kunci: gangguan pendengaran, pajanan kebisingan, usia, masa kerja, pekerja industri

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Exposure to noise is the most common cause of hearing loss, leading to noise induced hearing loss (NIHL). This study evaluated hearing loss associated with noise exposure related to age, length of employment, length of exposure, the use of personal protective equipment, smoking habits, hobbies associated noise and diabetes mellitus, hyperlipidemia and hypertension in workers. This is a cross-sectional observational study examined the independent variable, the dependent variable, and confounding variables at the same time. Using the company secondary data, through observation, measurement and questionnaire. Noise measurement results indicate that the potential area of potential noise is 63 dBA as the lowest noise and the highest is 110, 6 dBA, field noise level area ranging from 84.88 - 93 dBA. Nonfield noise area 79.5 dBA. Exposure effective noise below 80 dBA, either in the field or nonfield area; 7.1% of workers worked > 20 years, working life > 20 years, the hearing loss of workers 5.6%, workers aged > 40 years 40 is 5%. 42.9% of workers have a smoking habit, not found a relationship between smoking behavior with hearing loss. HPD consumption levels in workers earned as much as 90.5% of the workers who always wear APT, there is no relationship between the use of HPD with hearing loss. There were no relationship between hobby with hearing loss. As well as no relationship found between workers health status such as lipid profile (total cholesterol, HDL, LDL, and triglycerides), worker glucose blood levels and blood pressure with hearing loss.
Keywords: hearing loss, noise exposure, age, years of service, industry workers

Abstrak: Pekerja kontraktor bahan kimia di perusahaan minyak dan gas bumi PT. XYZ merupakan populasi berisiko terhadap pajanan Benzena disebabkan oleh aktifitas dan kondisi lingkungan kerja yang memungkinkan terpajan oleh uap Benzena. Penelitian ini bertujuan untuk memperkirakan tingkat risiko nonkarsinogenik dan karsinogenik disertai dengan analisis kemungkinan ketidaknormalan kadar darah akibat pajanan Benzena, untuk kemudian ditentukan manajemen risiko yang harus dilakukan. Penelitian merupakan studi potong lintang dilakukan terhadap seluruh pekerja kontraktor bahan kimia di PT. XYZ yang berjumlah 22 orang ditambah dengan 22 orang sebagai pembanding dipilih dari karyawan perusahaan PT. XYZ pada lokasi yang sama.
Hasil penelitian menunjukkan bahwa populasi pekerja bahan kimia di PT. XYZ berisiko terhadap pajanan Benzena nonkarsinogenik (RQ = 1,7442) dan karsinogenik (ECR = 1,76 x 10-4) pada durasi pajanan lifetime. Diketahui hubungan yang bermakna antara pajanan Benzena terhadap normalitas kadar hemoglobin (p = 0,015) dan eritrosit (p = 0,000). Risiko ketidaknormalan kadar hemoglobin dan eritrosit berturut-turut pada populasi terpajan adalah 6,92 kali (95% CI:1,28?37,29) dan 21,53 kali (95% CI:4,46?103,90) dibandingkan populasi tidak terpajan. Selain itu juga diketahui hubungan yang signifikan antara kenaikan jumlah asupan Benzena terhadap penurunan kadar haemoglobin (rs = -0,433; p = 0,044) dan eritrosit (rs = -0,474; p = 0,026).
Disimpulkan bahwa risiko kesehatan nonkarsinogenik dan karsinogenik akibat pajanan Benzena pada populasi pekerja bahan kimia di perusahaan minyak dan gas PT. XYZ akan terjadi pada durasi pajanan lifetime. Terdapat hubungan antara pajanan Benzena dengan ketidaknormalan hemoglobin dan eritrosit.

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Chemical contractor worker at the oil and gas company PT. XYZ is a population at risk to Benzene exposure due to its activities and work environment condition that possibly exposed by Benzene vapour. This research is aimed to estimate noncarsinogenic and carsinogenic risk level, complemented with blood counts abnormality analysisdue to Benzene exposure, then determining risk management shall be done. The research is cross sectional study was done to all chemical contractor worker at PT. XYZ, consist of 22 person, and additional 22 person as a control was selected from employee of PT. XYZ working at the same location. The research yield that chemical worker population at PT. XYZ is at risk to the noncarsinogenic (RQ = 1.7442) and carsinogenic (ECR = 1.76 x 10-4) Benzene exposure at the lifetime exposure duration.
Its known that there is a correlation between Benzene exposure with normality of haemoglobin (p = 0.015) and erythrocytes (p = 0.000). The risk of abnormality haemoglobin and erythrocytes counts is 6.92 times (95% CI:1.28?37.29) dan 21.53 times (95% CI:4.46?103.90) respectively compare to the non exposed population. In addition, its identified that there is a significant correlation between increased Benzene intake to the haemoglobin (rs = -0.433; p = 0.044) and erythrocytes (rs = -0.474; p = 0.026) counts reduction.
In summary noncarcinogenic and carcinogenic health risk due to Benzene exposure in the population of chemical worker at the oil and gas company PT. XYZ will occure at the lifetime exposure duration. There is a correlation between Benzene exposure with abnormality of haemoglobin and erythrocytes.

Abstrak: